Concluded Case

DAI WITH HYPOXIC BRAIN DAMAGE

8yrs/M presented to casualty today morning in unconscious and gasping condition with H/o fall from a single storey building directly on a concrete following LOC.Patient presented to ER with GCS 4/15 and Pupils were B/l 7mm SRTL.in Evening GCS was E1VetM1.NEUROSURGEON EXPLAINED POOR PROGNOSIS TO RELATIVES.KINDLY INTERPRET CT SCAN HEAD?

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Concluded answer
Diffuse axonal injury. Ventricles are not clearly visible. Gyri and sulci are also not clearly demarcated. Repeat CT after 24 hrs. Conservative management.
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Diffuse cerebral oedema following cerebo vascular truma. Common causes are head trauma, high altitude, malignant hypertension and anoxia. Localized cerebral edema: Occurs with any lesion of brain like tumor. Cerebral edema can result from brain trauma or from nontraumatic causes such as ischemic stroke, cancer, or brain inflammation due to meningitis or encephalitis. Vasogenic edema caused by amyloid-modifying treatments, such as monoclonal antibodies, is known as ARIA-E (amyloid-related imaging abnormalities edema).
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Diffuse axonal injury. Ventricles are not clearly visible. Gyri and sulci are also not clearly demarcated. Repeat CT after 24 hrs. Conservative management.
diffuse axonal injury cerebral oedema
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Global injury
Non - prominence of sulci and gyri - barely visible ventricles indicative of diffuse global axonal injury with severe compromise in Oxygen supply to brain
Sulci and gyri are not seen. Ventricles are compressed. No gray while matter difference . Anixic brain damage.
CT scan NCCT head -Normal. Concussion injury brain. Conversion reaction.
Diffuse Cerebral Edema due to traumatic brain injury
Ventilation, mannitol infusion.
Agreed with Dr.Venkatesh Venky.
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