Patient came to me with complain of brittle ant lower teeth . According to patient last year she got herpez zoster w.r.t.lower right region of face including tongue. After that teeth become brittle and due to brittleness 43 broken. What is d diagnosis and treatment?
Doc...what is the age of the patient... herpes zoster would not cause brittleness of teeth...pic shows poor oral hygiene.... and severe calculus and decay of teeth..... and what is the chief complaint of the patient... treatment depends on the chief complaint first and later others like scaling and restorations and all
Because of wrong method of doing brushing will Leeds to attrition of teeth . The patient food habits also we have to consider. The herpes simplex will not effect hard enamel r dentin . Because of wrong method of brushing attrition in the lower incisors and little canine areas . So smooth brush is recommended in this case and better pt can apply sensoform r sensodin r cavison paste for desensitization of front incisors and all other attritioned teeth in the oral cavity , which r not visible . We can cheque there may be any epidemic history of any fluorosis . In peptic ulcer and dispepsia in these cases also there will be severe etching in the lingual side due to regurgitation of gastric juice which contains hydrochloric acid . Educate pt how to do brushing on hopelessly attritioned teeth and any Bruksim nocturnal and day time of grinding of teeth . Please take full history of pt
Enamel hypoplasia or flourosis Conservative treatment and prosthesis for missing teeth
As u know this is not possible by herpes zoster. May be poor oral hygiene during the period of illness caused severe calculus deposits or decay. I believe prosthesis in this case is appropriate
THIS brittleness is not due to herpes zoster inf. All ant teeth cervical caries brown discoloration. Adv blood --TC /DC ESR 2 HRS. PT by cast Muslim??
Check whether ur patient is chronic gastritis patient with reflux oseophagitis ... Probably caused by herpes zoster or drug induced ..Or bulimia patient .. Looks more of erosion to me ..treatment is full moth rehabilitation with treating underlying systemic cause ..
enamel hypoplasia... vigorous brushing worsen the condition.
Poor oral hygiene... Discoloration calculus n decayed teeth T/t- Scaling n restoration Herpes zooster not a cause of brittle teeth...acrdng to me it may be due to GERD or vit d deficiency so Adv CBC Vit D Tab Limcee 500mg bd Shelcal 500mg od. Pan d od ×15 days
Enamel hypoplasia Flourosis Prosthesis
I think its only coz of poor oral hygiene
Cases that would interest you
- Login to View the image
GASTROINTESTINAL MANIFESTATIONS OF SYSTEMIC LUPUS ERYTHEMATOSUS: Gastrointestinal complications are common in SLE. ORAL CAVITY : Mucous membrane involvement occurs in 12-45 %of patients with SLE.Oral or nasopharyngeal ulceration are usually painless and is one of the eleven diagnostic criteria of SLE.The ulcers are discoid or ulcerative DISCOID lesions are painful,characterized by erythema,atrophy and depigmentation and usually occurs on lips. ULCERS are punched out erosions with surrounding erythema,irregularly shaped raised white plaques tend to occur on hard or soft palate and buccal mucosa.These are usually painless. TREATMENT of oral lesions: *Topical glucocorticoids. *Tacrolimus ointment. *Intralesional corticosteroids. *Systemic antimalarial drugs. OESOPHAGUS : DYSPHAGIA,DYSMOTILITY,VASCULITIS, ISCHEMIA,ULCERATION,PERFORATION. DYSPHAGIA:Dysphagia occurs due to SLE or to complications associated with therapy such as pill oesophagitis and oral candidiasis DYSMOTILITY :Hypotensive LES(lower esophageal sphincter) and aperistalysis are documented by manometry. VASCULITIS-ISCHEMIA-ULCERATION- PERFORATION :Ischemia from vasculitis can lead to esophageal ulceration and perforation. TREATMENT : *Hypomotility is treated with metaclopramide. *GERD symptoms are treated with routine GERD precautions and proton pump inhibitors. *Immunomodulatory agents are used for treating SLE,but they can cause opportunistic oesophagitis from candidiasis,cytomegalovirus and herpes simplex virus. STOMACH &DUODENUM : DYSPEPSIA,PEPTIC ULCER DISEASE AND GASTRIC ANTRAL VASCULAR ECTASIA (GAVE) DYSPEPSIA is due to treatment with NSAID's or corticosteroids. PEPTIC ULCER DISEASE is also due to NSAID's and corticosteroids.Therefore prophylactic therapy for peptic ulcer is beneficial in SLE patients with dyspepsia,history of peptic ulcer disease,and on therapy with NSAID's and corticosteroids. GAVE :GAVE is an erosive type of atrophic gastritis characterized by marked veno-capillary ectasia.This causes chronic gastro- intestinal bleeding or irondeficiencyanemia. Dilated small blood vessels cause streaky long red areas that resemble the markings on watermelon when viewed endoscopically SO CALLED AS WATERMELON STOMACH. SMALL &LARGE BOWEL : ABDOMINAL PAIN,GASTROINTESTINAL BLEEDING,DYSMOTILITY,DIARRHEA,PROTEIN LOOSING ENTEROPATHY ,PNEUMATOSIS CYSTOIDES INTESTINALIS ,RECTAL ULCERS, STEATORRHOEA,PANCREATITIS, ACALCULUS CHOLECYSTITIS : ABDOMINAL PAIN : can be caused by mesenteric vasculitis and infarction,acalculous cholecystitis,peptic ulcer disease and pancreatitis. IINTESTINAL VASCULITIS. causes transmural inflammation with fibrinoid necrosis.It is most commonly found in terminal ileum and cecum where the blood supply is from superior mesenteric artery.Endoscopically,the bowel appears thickened and friable with ulceration and submucosal hemorrhages. COLON focal /diffuse ulcers,ischemic colitis or colonic diverticula. PROTEIN LOOSING ENTEROPATHY (PLE) This is characterized by significant hypo- albuminenia and diarrhea without proteinuria These are a result of villous atrophy with inflammatory. infiltrates and submucosal edema.Disease activity is measured by monitoring fecal excretion of radiolabelled albumin. PNEUMATOSIS CYSTOIDES INTESTINALIS PCI is gas filled cysts in the submucosa or subserosa of bowel wall.This radiological sign is suggestive of necrotising enterocolitis STEATORRHEA :occurs secondary to villus blunting and immune complex deposition.
Dr. Suvarchala Pratap8 Likes6 Answers - Login to View the image
41 year-old male presented to with the sudden onset of severe epigastric pain. He had a history of heart burn and dyspeptic symptoms for past 10 years. On physical exam, he had a temperature of 101.4°F, a pulse of 118 and a blood pressure of 128/72. Abdomen was tender with board like rigidity.
Jithin Prakash1 Like19 Answers - Login to View the image
Diagnosis of all the UGI Endoscopic findings please..?
Dr. Shankar Sastry10 Likes27 Answers - Login to View the image
Smoker , ex alcoholic 45 yrs patient presented with epigastric and retro sternal burning since 5 days early in the morning 2-5 am and subsides by its own after 6 am ... he is normatensive and non diabetic pls help me in dx and mngmnt
Dr. Shivakumar Kongutte0 Like28 Answers - Login to View the image
9yrs old female kid surfing with reddish patches, burning sensation, with inflammation over back and chest region's since 3days and increasing day by day. No h/o insect bite, Please diagnose it and suggest proper treatment for it. @Dr. Navoday Raju @Dr. Degulmdi Devendrappa @Dr. Kamalakar Puripanda
Dr. Mahesh Raj6 Likes76 Answers
7 Likes